[Effects of L-carnitine and bisoprolol on endoplasmic reticulum stress-mediated myocardial injury after cardiopulmonary resuscitation in rats]

Zhonghua Yi Xue Za Zhi. 2015 May 19;95(19):1475-8.
[Article in Chinese]

Abstract

Objective: To explore the effects of L-carnitine and bisoprolol on endoplasmic reticulum stress-mediated myocardial injury after cardiopulmonary resuscitation in rats.

Methods: A total of 75 Sprague-Dawley rats were randomly divided into 5 groups of sham operation (sham); cardiopulmonary resuscitation (CR), L-carnitine (L), bisoprolol treatment (B) and L-carnitine and bisoprolol treatment (LB). Myocardial pathological changes were detected by hematoxylin and eosin, myocardial apoptosis by terminal deoxynucleotidyl transferase mediated dUTP nick and the expressions of key factors in endoplasmic reticulum (ER) by Western blot.

Results: Compared with sham group, apoptosis and pathological lesions significantly increased in other groups (P < 0.05). And the levels of GRP78, CHOP and Caspase-12 were significantly higher (P < 0.05). The expression of ER factor protein and the degrees of myocardial injury in LB and B groups decreased compared with CR group. And LB group was the most obvious.

Conclusions: Combined use of levocarnitine and bisoprolol exerts protective effects on cardiopulmonary resuscitation in rats. And the mechanism may be related to an inhibition of ER stress.

MeSH terms

  • Animals
  • Apoptosis
  • Bisoprolol
  • Cardiopulmonary Resuscitation
  • Carnitine
  • Caspase 12
  • Endoplasmic Reticulum
  • Endoplasmic Reticulum Stress*
  • Heart Injuries*
  • Heat-Shock Proteins
  • Lung
  • Myocardium
  • Rats
  • Rats, Sprague-Dawley

Substances

  • GRP78 protein, rat
  • Heat-Shock Proteins
  • Casp12 protein, rat
  • Caspase 12
  • Carnitine
  • Bisoprolol