Abstract
Disposal of damaged mitochondria is tightly controlled by auto-inhibition mechanisms that keep the ubiquitin ligase Parkin in check. Several new structural studies provide insight into how PINK1-dependent phosphorylation of ubiquitin and Parkin may progressively relieve Parkin auto-inhibition.
MeSH terms
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Enzyme Activation / genetics*
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Humans
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Mitochondria / metabolism*
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Models, Biological*
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Models, Molecular*
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Protein Kinases / chemistry*
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Protein Kinases / metabolism*
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Ubiquitin-Protein Ligases / chemistry*
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Ubiquitin-Protein Ligases / metabolism*
Substances
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Ubiquitin-Protein Ligases
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Protein Kinases