GLUT4 translocation is not impaired after acute exercise in skeletal muscle of women with obesity and polycystic ovary syndrome

Obesity (Silver Spring). 2015 Nov;23(11):2207-15. doi: 10.1002/oby.21217. Epub 2015 Sep 16.

Abstract

Objective: The aim of this study was to examine the effects of acute exercise on insulin signaling in skeletal muscle of women with polycystic ovary syndrome (PCOS) and controls (CTRL).

Methods: Fifteen women with obesity and PCOS and 12 body mass index-matched CTRL participated in this study. Subjects performed a 40-min single bout of exercise. Muscle biopsies were performed before and 60 min after exercise. Selected proteins were assessed by Western blotting.

Results: CTRL, but not PCOS, showed a significant increase in PI3-k p85 and AS160 Thr 642 after a single bout of exercise (P = 0.018 and P = 0.018, respectively). Only PCOS showed an increase in Akt Thr 308 and AMPK phosphorylation after exercise (P = 0.018 and P = 0.018, respectively). Total GLUT4 expression was comparable between groups (P > 0.05). GLUT4 translocation tended to be significantly higher in both groups after exercise (PCOS: P = 0.093; CTRL: P = 0.091), with no significant difference between them (P > 0.05).

Conclusions: A single bout of exercise elicited similar GLUT4 translocation in skeletal muscle of PCOS and CTRL, despite a slightly differential pattern of protein phosphorylation. The absence of impairment in GLUT4 translocation suggests that PCOS patients with obesity and insulin resistance may benefit from exercise training.

Publication types

  • Controlled Clinical Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Body Mass Index
  • Exercise / physiology*
  • Female
  • Glucose Transporter Type 4 / metabolism*
  • Humans
  • Insulin / metabolism
  • Insulin Resistance
  • Muscle, Skeletal / metabolism*
  • Obesity / complications
  • Obesity / metabolism*
  • Phosphorylation
  • Polycystic Ovary Syndrome / complications
  • Polycystic Ovary Syndrome / metabolism*
  • Polycystic Ovary Syndrome / therapy*
  • Protein Transport
  • Signal Transduction / drug effects
  • Young Adult

Substances

  • Glucose Transporter Type 4
  • Insulin
  • SLC2A4 protein, human