[Nuclear factor I-C inhibits platelet-derived growth factor-induced enhancement of dermal fibroblast sensitivity to TGF-β]

Nan Fang Yi Ke Da Xue Xue Bao. 2015 Aug;35(9):1245-50.
[Article in Chinese]

Abstract

Objective: To investigate the effect of nuclear factor I-C (NFI-C) on platelet-derived growth factor (PDGF)-induced up-regulation of TGF-β receptor II (TβRII) in dermal fibroblasts.

Methods: A lentiviral vector containing NFI-C sequence (Lenti-GFP-NFI-C) was transfected into a human foreskin fibroblast cell line (HFF-1). Cultured HFF-1 cells, cells transfected with Lenti-GFP-NFI-C, and cells transfected with a negative virus were stimulated with PDGF-BB, and Western blotting and RT-qPCR were used to detect the expression levels of TβRII in the treated cells.

Results: PDGF treatment significantly increased the expression level of TβRII in HFF-1 cells (P<0.05). The cells transfected with Lenti-GFP-NFI-C expressed a significantly lower level of TβRII than non-transfected cells in response to PDGF stimulation (P<0.05), but the negative virus showed no such inhibitory effect (P>0.05). No significant difference was found in the expression level of TβRII protein between cells transfected with Lenti-GFP-NFI-C-transfection before PDGF stimulation and the blank control cells.

Conclusion: NFI-C can inhibit PDGF-induced up-regulation of TβRII and thus reduce the sensitivity of the dermal fibroblasts to TGF-β.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Becaplermin
  • Cell Line
  • Fibroblasts / drug effects*
  • Genetic Vectors
  • Humans
  • Lentivirus
  • NFI Transcription Factors / genetics*
  • Platelet-Derived Growth Factor / pharmacology*
  • Protein Serine-Threonine Kinases / metabolism
  • Proto-Oncogene Proteins c-sis
  • Receptor, Transforming Growth Factor-beta Type II
  • Receptors, Transforming Growth Factor beta / metabolism
  • Transfection
  • Transforming Growth Factor beta / pharmacology*
  • Up-Regulation

Substances

  • NFI Transcription Factors
  • NFIC protein, human
  • Platelet-Derived Growth Factor
  • Proto-Oncogene Proteins c-sis
  • Receptors, Transforming Growth Factor beta
  • Transforming Growth Factor beta
  • Becaplermin
  • Protein Serine-Threonine Kinases
  • Receptor, Transforming Growth Factor-beta Type II