Bone marrow-derived mesenchymal stem cell-secreted IL-8 promotes the angiogenesis and growth of colorectal cancer

Oncotarget. 2015 Dec 15;6(40):42825-37. doi: 10.18632/oncotarget.5739.

Abstract

Mesenchymal stem cells (MSCs) have recently been shown to home to tumors and contribute to the formation of the tumor-associated stroma. In addition, MSCs can secrete paracrine factors to facilitate tumor progression. However, the involvement of MSC-derived cytokines in colorectal cancer (CRC) angiogenesis and growth has not been clearly addressed. In this study, we report that interleukin-8 (IL-8) was the most highly upregulated pro-angiogenic factor in MSCs co-cultured with CRC cells and was expressed at substantially higher levels in MSCs than CRC cells. To evaluate the effect of MSC-derived IL-8 on CRC angiogenesis and growth, we used MSCs that expressed small hairpin (interfering) RNAs (shRNA) targeting IL-8 (shIL-8-MSCs). We found that MSC-secreted IL-8 promoted human umbilical vein endothelial cell (HUVEC) proliferation and migration, tube-formation ability and CRC cell proliferation. Additionally, in vivo studies showed that MSCs promoted tumor angiogenesis partially through IL-8. Taken together, these findings suggest that IL-8 secreted by MSCs promotes CRC angiogenesis and growth and can therefore serve as a potential novel therapeutic target.

Keywords: angiogenesis; colorectal cancer; interleukin-8; mesenchymal stem cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Bone Marrow Cells / metabolism
  • Cell Proliferation / drug effects
  • Cells, Cultured
  • Coculture Techniques
  • Colorectal Neoplasms / metabolism
  • Colorectal Neoplasms / pathology*
  • Culture Media, Conditioned / pharmacology
  • Enzyme-Linked Immunosorbent Assay
  • Heterografts
  • Human Umbilical Vein Endothelial Cells / drug effects
  • Humans
  • Immunohistochemistry
  • Interleukin-8 / metabolism*
  • Mesenchymal Stem Cells / metabolism*
  • Mice
  • Mice, Nude
  • Neovascularization, Pathologic / metabolism*
  • Neovascularization, Pathologic / pathology
  • RNA, Small Interfering
  • Reverse Transcriptase Polymerase Chain Reaction
  • Transfection

Substances

  • CXCL8 protein, human
  • Culture Media, Conditioned
  • Interleukin-8
  • RNA, Small Interfering