Nuclear staining of fgfr-2/stat-5 and runx-2 in mucinous breast cancer

Exp Mol Pathol. 2016 Feb;100(1):39-44. doi: 10.1016/j.yexmp.2015.11.003. Epub 2015 Nov 6.

Abstract

Mucinous carcinoma (MBC) is a rare subtype of breast cancer characterized by the production of variable amounts of mucin, with a prognosis better than that of non-mucinous carcinomas (NMBC). The aim of this project was to evaluate the expression of STAT-5, RUNX-2, and FGFR-2 in a cohort of MBC and compare it with that of NMBC using standard immunohistochemistry. STAT-5 and RUNX-2 are two transcription factors with cytoplasmic and/or nuclear localization that have been related to FGFR-2, a tyrosine kinase growth factor receptor that can interact with STAT-5 and with PR in the nuclei of breast cancer cells. Membranous, cytoplasmic, and nuclear staining were evaluated and expressed as the percentage of stained cells (0-100%) multiplied by the staining intensity (0-3), thus obtaining an index ranging from 0 to 300. Nuclear and/or cytoplasmic immunoreactivity of the three proteins were detected in a high number of NMBC. Nuclear FGFR-2 staining correlated with nuclear STAT-5 (p<0.05) and nuclear RUNX-2 (p<0.01) in both tumor types; however MBC had a significant higher expression of nuclear FGFR-2 (p<0.01) and RUNX-2 (p<0.05) than that of NMBC, and displayed positive immunoreactivity of the 3 proteins in 70.8% of the cases. These results suggest that these proteins may have a role in the progression of the mucinous phenotype, in which nuclear STAT-5 may inhibit RUNX-2 prometastatic effect.

Keywords: Breast cancer; FGFR-2; Mucinous breast carcinoma; RUNX-2; STAT-5.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenocarcinoma, Mucinous / metabolism
  • Adenocarcinoma, Mucinous / pathology
  • Breast Neoplasms / metabolism*
  • Breast Neoplasms / pathology
  • Carcinoma, Ductal, Breast / diagnosis*
  • Carcinoma, Ductal, Breast / metabolism*
  • Cell Nucleus / metabolism
  • Core Binding Factor Alpha 1 Subunit / metabolism*
  • Female
  • Humans
  • Immunohistochemistry / methods
  • Prognosis
  • Receptor, Fibroblast Growth Factor, Type 2 / metabolism*
  • STAT5 Transcription Factor / metabolism*

Substances

  • Core Binding Factor Alpha 1 Subunit
  • RUNX2 protein, human
  • STAT5 Transcription Factor
  • FGFR2 protein, human
  • Receptor, Fibroblast Growth Factor, Type 2