[G14453A mutation in mitochondrial myopathy encephalomyopathy with lactic acidosis and stroke-like episodes]

Zhonghua Yi Xue Za Zhi. 2015 Aug 25;95(32):2623-5.
[Article in Chinese]

Abstract

Objective: To analyz mitochondrial DNA mutation in one case of mitochondrial myopathy, encephalopathy, lactic acidosis and stroke-like episodes (MELAS).

Methods: The patient, a 10-years-old boy,clinically diagnosed as MELAS. The clinical information was collected, and the normal mitochondrial mutations (such as A3243G, A8344G, T8993G/C, G13513A etc) were excluded. PCR-sequencing was used to analyz the whole-mitochondrial genome (16.6 kb), and PCR-RFLP was used to confirm the mutations.

Results: m.G14453A mutation was detected from the patient's peripheral blood and urine, but it was not found in his parents and 100 normal controls. The m.G14453A mutation was confirmed by PCR-RFLP, and mutation ratio of in blood was 56.8% and urine was 72.5%. The activity of complex I was decreased (67.6 nmol·min⁻¹·mg⁻¹). This was the first report that m.G14453A mutation could lead to MELAS in China according www.mitomap.org web and NCBI.

Conclusion: m.G14453A mutation is one of the causative mutations in MELAS, but the mechanism of the mutation should be studied in future.

Publication types

  • Case Reports

MeSH terms

  • Child
  • China
  • DNA, Mitochondrial
  • Diagnosis, Differential
  • Genome, Mitochondrial
  • Humans
  • MELAS Syndrome*
  • Male
  • Mutation*
  • Polymerase Chain Reaction
  • Polymorphism, Restriction Fragment Length*
  • Stroke*

Substances

  • DNA, Mitochondrial