Lineage-specific requirement of c-abl function in normal hematopoiesis

Science. 1989 Sep 8;245(4922):1107-10. doi: 10.1126/science.2672339.

Abstract

Structural abnormalities of the c-abl proto-oncogene are found in hematopoietic cells of more than 90 percent of individuals with chronic myelogenous leukemia. Therefore c-abl may be important in normal as well as malignant hematopoiesis. Normal human hematopoietic progenitor cells were exposed to three different c-abl sense or antisense oligodeoxynucleotides, and the effects on myeloid and erythroid colony formation were examined. The c-abl antisense oligodeoxynucleotides inhibited myeloid, but not erythroid, colony formation. The c-abl sense oligodeoxynucleotides and bcr sense and antisense oligodeoxynucleotides were not inhibitory in this assay. These data show that c-abl is critical in normal myelopoiesis and may explain the relatively selective expansion of leukocytes in patients with chronic myelogenous leukemia.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Blood Cells / drug effects
  • Blood Cells / metabolism
  • Blood Cells / physiology
  • Bone Marrow
  • Cell Differentiation / drug effects
  • Colony-Forming Units Assay
  • Erythropoiesis / drug effects
  • Hematopoiesis* / drug effects
  • Hematopoietic Stem Cells / drug effects
  • Hematopoietic Stem Cells / metabolism
  • Hematopoietic Stem Cells / physiology
  • Humans
  • Oligonucleotides / pharmacology
  • Oligonucleotides, Antisense
  • Proto-Oncogene Mas
  • Proto-Oncogene Proteins / biosynthesis
  • Proto-Oncogenes*

Substances

  • MAS1 protein, human
  • Oligonucleotides
  • Oligonucleotides, Antisense
  • Proto-Oncogene Mas
  • Proto-Oncogene Proteins