The biology behind interleukin-6 targeted interventions

Curr Opin Rheumatol. 2016 Mar;28(2):152-60. doi: 10.1097/BOR.0000000000000255.

Abstract

Purpose of review: Biological drugs that target the inflammatory cytokine interleukin-6 (IL-6) are increasingly considered as therapies for chronic disease and cancer. The purpose of this review is to place the biology of IL-6 in context. Here, we provide information on the biology behind IL-6 and consider mechanisms that are relevant to the application of IL-6 targeted therapies.

Recent findings: The clinical blockade of IL-6 activity with tocilizumab has fuelled considerable interest in the biology behind this inflammatory cytokine. Although IL-6 impacts both innate and adaptive immunity, and the control of tissue homeostasis, the signalling mechanisms that control IL-6 responsiveness are complex. Several alternative IL-6-directed interventions with unique modes of action are now approaching the clinic. However, various questions still remain about how and when to block IL-6. Owing to the complexity of IL-6 biology, this is not trivial. In this review, we introduce the immunobiology of IL-6 and explore the different therapeutic strategies in development that inhibit IL-6 activity.

Summary: Various inhibitors of IL-6 bioactivity are now in development of routine clinical practice. The key is to understand how best to apply these drugs. This review provides useful insight into the workings of IL-6 in health and disease.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Antibodies, Monoclonal, Humanized / pharmacology
  • Antibodies, Monoclonal, Humanized / therapeutic use
  • Cytokine Receptor gp130 / metabolism
  • Humans
  • Interleukin-6 / antagonists & inhibitors
  • Interleukin-6 / immunology*
  • Molecular Targeted Therapy / methods
  • Signal Transduction / immunology

Substances

  • Antibodies, Monoclonal, Humanized
  • IL6 protein, human
  • IL6ST protein, human
  • Interleukin-6
  • Cytokine Receptor gp130
  • tocilizumab