Serum elevation of B lymphocyte stimulator does not increase regulatory B cells in glioblastoma patients undergoing immunotherapy

Cancer Immunol Immunother. 2016 Feb;65(2):205-11. doi: 10.1007/s00262-015-1784-3. Epub 2016 Jan 12.

Abstract

Regulatory B cells that secrete IL-10 (IL-10(+) Bregs) represent a suppressive subset of the B cell compartment with prominent anti-inflammatory capacity, capable of suppressing cellular and humoral responses to cancer and vaccines. B lymphocyte stimulator (BLyS) is a key regulatory molecule in IL-10(+) Breg biology with tightly controlled serum levels. However, BLyS levels can be drastically altered upon chemotherapeutic intervention. We have previously shown that serum BLyS levels are elevated, and directly associated, with increased antigen-specific antibody titers in patients with glioblastoma (GBM) undergoing lymphodepletive temozolomide chemotherapy and vaccination. In this study, we examined corresponding IL-10(+) Breg responses within this patient population and demonstrate that the IL-10(+) Breg compartment remains constant before and after administration of the vaccine, despite elevated BLyS levels in circulation. IL-10(+) Breg frequencies were not associated with serum BLyS levels, and ex vivo stimulation with a physiologically relevant concentration of BLyS did not increase IL-10(+) Breg frequency. However, BLyS stimulation did increase the frequency of the overall B cell compartment and promoted B cell proliferation upon B cell receptor engagement. Therefore, using BLyS as an adjuvant with therapeutic peptide vaccination could promote humoral immunity with no increase in immunosuppressive IL-10(+) Bregs. These results have implications for modulating humoral responses in human peptide vaccine trials in patients with GBM.

Keywords: BAFF; BLyS; Bregs; GBM; Immunotherapy; Lymphopenia.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Antibodies / blood
  • Antibodies / immunology
  • Antineoplastic Agents, Alkylating / pharmacology
  • Antineoplastic Agents, Alkylating / therapeutic use
  • B-Cell Activating Factor / blood*
  • B-Lymphocytes, Regulatory / immunology*
  • B-Lymphocytes, Regulatory / metabolism
  • Cancer Vaccines / administration & dosage
  • Cancer Vaccines / immunology
  • Dacarbazine / analogs & derivatives
  • Dacarbazine / pharmacology
  • Dacarbazine / therapeutic use
  • ErbB Receptors / antagonists & inhibitors
  • ErbB Receptors / immunology
  • Glioblastoma / blood*
  • Glioblastoma / immunology*
  • Glioblastoma / mortality
  • Glioblastoma / therapy
  • Humans
  • Immunotherapy / methods
  • Interleukin-10 / metabolism
  • Lymphocyte Activation / immunology
  • Lymphocyte Count*
  • Temozolomide
  • Vaccines, Subunit / administration & dosage
  • Vaccines, Subunit / immunology

Substances

  • Antibodies
  • Antineoplastic Agents, Alkylating
  • B-Cell Activating Factor
  • Cancer Vaccines
  • Vaccines, Subunit
  • epidermal growth factor receptor VIII
  • Interleukin-10
  • Dacarbazine
  • ErbB Receptors
  • Temozolomide