H. pylori's BabA Embraces Change

Emily Goers Sweeney; Karen Guillemin

doi:10.1016/j.chom.2015.12.013

H. pylori's BabA Embraces Change

Cell Host Microbe. 2016 Jan 13;19(1):5-7. doi: 10.1016/j.chom.2015.12.013.

Authors

Emily Goers Sweeney¹, Karen Guillemin²

Affiliations

¹ Institute of Molecular Biology, University of Oregon, Eugene, OR 97403, USA.
² Institute of Molecular Biology, University of Oregon, Eugene, OR 97403, USA. Electronic address: [email protected].

PMID: 26764590
DOI: 10.1016/j.chom.2015.12.013

Abstract

Long before the cause was discovered, peptic ulcers were known to occur preferentially in individuals of certain blood groups. In this issue of Cell Host & Microbe, Moonens et al. (2016) perform structural analyses of the Helicobacter pylori adhesin BabA to determine how the bacteria discriminately bind different blood group glycans.

Publication types

Comment

MeSH terms

Adhesins, Bacterial / metabolism
Helicobacter Infections / microbiology*
Helicobacter pylori / metabolism*
Humans

Substances

Adhesins, Bacterial