E-cadherin maintains the activity of neural stem cells and inhibits the migration

Int J Clin Exp Pathol. 2015 Nov 1;8(11):14247-51. eCollection 2015.

Abstract

Cadherins are important adhesion molecules that mediate adhesions and communications between cells. These molecules participate in the formation and maintenance of multicellular organisms including the stem cells. E-cadherin is one of the classic cadherins which is reported to be essential for the survival and self-renewal of embryonic stem cells. Moreover, it could induce cell proliferation inhibitory signaling to regulate cell proliferation. In our study, we over-expressed and silenced E-cadherin in NSCs by lentiviral ways. Transgenic cells were confirmed by both quantitative RT-PCR and western blot. Results of MTT assay showed that over-expression of E-cadherin could enhance the cell activity. Furthermore, we performed Transwell chamber assay to analyze its role in regulation of cell migration. The results showed that the migration percent of over-expression cells was lower than control. Our results indicated that E-caherin would maintain the stemness of NSCs and reduce cells migration.

Keywords: Cadherins; E-cadherin; N-cadherin; cell activity; cell migration; neural stem cell.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cadherins / genetics
  • Cadherins / metabolism*
  • Cell Movement*
  • Cell Proliferation*
  • Cells, Cultured
  • Genetic Vectors
  • Lentivirus / genetics
  • Mice
  • Neural Stem Cells / metabolism*
  • Phenotype
  • RNA Interference
  • Signal Transduction
  • Time Factors
  • Transfection

Substances

  • Cadherins
  • Cdh1 protein, mouse
  • Cdh2 protein, mouse