Curcumin inhibits the proliferation and invasion of human osteosarcoma cell line MG-63 by regulating miR-138

Int J Clin Exp Pathol. 2015 Nov 1;8(11):14946-52. eCollection 2015.

Abstract

Objective: In this study, we screened the different human osteosarcoma cell line MG-63 miRNAs after the treatment of curcumin and explored the effects of curcumin on MG-63 cells and its mechanism.

Methods: Affemitrix miRNA chip was used to detect the changes of miRNA expression profile in MG-63 cells before and after curcumin treatment, and screen different expression of miRNAs. The target gene of miRNA was analyzed by bioinformatics. The expression levels of miRNA-138 target genes Smad4, NFκB p65 and cyclin D3 were detected. MTT and Transwell Cell invasion assays were used to observe the effects of curcumin on MG-63 cells.

Results: Curcumin could significantly inhibit the proliferation of MG-63 cells and the expression levels of miRNA-138 target genes Smad4, NFκB p65 and cyclin D3 in MG-63 cells (P<0.05); overexpression of hsa-miR-138 down-regulated the expression levels of Smad4, NFκB p65 and cyclin D3 compared with the treatment of curcumin, while inhibition of hsa-miR-138 up-regulated the expression levels of Smad4, NFκB p65 and cyclin D3.

Conclusions: Curcumin could increase the expression of hsa-miR-138, hsa-miR-138 inhibited cell proliferation and invasive ability by inhibition of its target genes.

Keywords: Curcumin; MG-63 cells; invasion; miR-138; miRNAs; proliferation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antineoplastic Agents / pharmacology*
  • Blotting, Western
  • Bone Neoplasms / pathology*
  • Cell Line, Tumor
  • Cell Movement / drug effects
  • Cell Movement / genetics
  • Cell Proliferation / drug effects
  • Cell Proliferation / genetics
  • Curcumin / pharmacology*
  • Gene Expression Regulation, Neoplastic / drug effects*
  • Humans
  • MicroRNAs / biosynthesis*
  • Oligonucleotide Array Sequence Analysis
  • Osteosarcoma / pathology*
  • Real-Time Polymerase Chain Reaction
  • Transfection

Substances

  • Antineoplastic Agents
  • MIRN138 microRNA, human
  • MicroRNAs
  • Curcumin