A novel genomic alteration of LSAMP associates with aggressive prostate cancer in African American men

EBioMedicine. 2015 Oct 31;2(12):1957-64. doi: 10.1016/j.ebiom.2015.10.028. eCollection 2015 Dec.

Abstract

Evaluation of cancer genomes in global context is of great interest in light of changing ethnic distribution of the world population. We focused our study on men of African ancestry because of their disproportionately higher rate of prostate cancer (CaP) incidence and mortality. We present a systematic whole genome analyses, revealing alterations that differentiate African American (AA) and Caucasian American (CA) CaP genomes. We discovered a recurrent deletion on chromosome 3q13.31 centering on the LSAMP locus that was prevalent in tumors from AA men (cumulative analyses of 435 patients: whole genome sequence, 14; FISH evaluations, 101; and SNP array, 320 patients). Notably, carriers of this deletion experienced more rapid disease progression. In contrast, PTEN and ERG common driver alterations in CaP were significantly lower in AA prostate tumors compared to prostate tumors from CA. Moreover, the frequency of inter-chromosomal rearrangements was significantly higher in AA than CA tumors. These findings reveal differentially distributed somatic mutations in CaP across ancestral groups, which have implications for precision medicine strategies.

Keywords: African American; ERG; Genome; LSAMP; PTEN; Prostate cancer.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Aged
  • Biomarkers, Tumor
  • Black or African American / genetics*
  • Cell Adhesion Molecules, Neuronal / genetics*
  • Cluster Analysis
  • Disease Progression
  • GPI-Linked Proteins / genetics
  • Gene Deletion
  • Gene Rearrangement
  • Genetic Association Studies*
  • Genetic Loci
  • Genetic Variation*
  • Genomics
  • High-Throughput Nucleotide Sequencing
  • Humans
  • Male
  • Middle Aged
  • Mutation
  • Neoplasm Grading
  • Neoplasm Staging
  • Oncogene Proteins, Fusion / genetics
  • PTEN Phosphohydrolase
  • Polymorphism, Single Nucleotide
  • Prostatic Neoplasms / genetics*
  • Prostatic Neoplasms / metabolism
  • Prostatic Neoplasms / pathology*
  • Reproducibility of Results

Substances

  • Biomarkers, Tumor
  • Cell Adhesion Molecules, Neuronal
  • GPI-Linked Proteins
  • Oncogene Proteins, Fusion
  • TMPRSS2-ERG fusion protein, human
  • limbic system-associated membrane protein
  • PTEN Phosphohydrolase
  • PTEN protein, human