Abstract
Methionine aminopeptidase 2 (MetAP2) is an enzyme that cleaves an N-terminal methionine residue from a number of newly synthesized proteins. Pre-clinical and clinical studies suggest that MetAP2 inhibitors could be used as a novel treatment for obesity. Herein we describe our use of fragment screening methods and structural biology to quickly identify and elaborate an indazole fragment into a series of reversible MetAP2 inhibitors with <10nM potency, excellent selectivity, and favorable in vitro safety profiles.
Keywords:
FBDD; Fragment-based drug discovery; Indazole; MetAP2; Metalloprotease; Methionine aminopeptidase 2.
Copyright © 2016 Elsevier Ltd. All rights reserved.
MeSH terms
-
Administration, Oral
-
Aminopeptidases / antagonists & inhibitors*
-
Aminopeptidases / metabolism
-
Animals
-
Body Weight / drug effects*
-
Dose-Response Relationship, Drug
-
Drug Discovery*
-
Enzyme Inhibitors / administration & dosage
-
Enzyme Inhibitors / chemistry
-
Enzyme Inhibitors / pharmacology*
-
Glycoproteins / antagonists & inhibitors*
-
Glycoproteins / metabolism
-
Humans
-
Indazoles / chemical synthesis
-
Indazoles / chemistry
-
Indazoles / pharmacology*
-
Methionyl Aminopeptidases
-
Mice
-
Mice, Obese
-
Models, Molecular
-
Molecular Structure
-
Obesity / drug therapy*
-
Structure-Activity Relationship
Substances
-
Enzyme Inhibitors
-
Glycoproteins
-
Indazoles
-
Aminopeptidases
-
METAP2 protein, human
-
Methionyl Aminopeptidases