The WNT-less wonder: WNT-independent β-catenin signaling

Pigment Cell Melanoma Res. 2016 Sep;29(5):524-40. doi: 10.1111/pcmr.12501. Epub 2016 Aug 1.

Abstract

β-catenin is known as an Armadillo protein that regulates gene expression following WNT pathway activation. However, WNT-independent pathways also activate β-catenin. During the establishment of the melanocyte lineage, β-catenin plays an important role. In the context of physiopathology, β-catenin is activated genetically or transiently in various cancers, including melanoma, where it can be found in the nucleus of tumors. In this review, we discuss alternative pathways that activate β-catenin independent of WNTs and highlight what is known regarding these pathways in melanoma. We also discuss the role of β-catenin as a transcriptional regulator in various cell types, with emphasis on the different transcription factors it associates with independent of WNT induction. Finally, the role of WNT-independent β-catenin in melanocyte development and melanomagenesis is also discussed.

Keywords: catenin; development; melanocyte; therapy.

Publication types

  • Review
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Gene Expression Regulation, Neoplastic*
  • Humans
  • Melanoma / metabolism
  • Melanoma / pathology*
  • Signal Transduction
  • Transcription Factors / metabolism*
  • Wnt Signaling Pathway
  • beta Catenin / metabolism*

Substances

  • Transcription Factors
  • beta Catenin