BRAT1-associated neurodegeneration: Intra-familial phenotypic differences in siblings

Am J Med Genet A. 2016 Nov;170(11):3033-3038. doi: 10.1002/ajmg.a.37853. Epub 2016 Aug 2.

Abstract

Recessive mutations in BRAT1 cause lethal neonatal rigidity and multifocal seizure syndrome, a phenotype characterized by neonatal microcephaly, hypertonia, and refractory epilepsy with premature death by age 2 years. Recently, attenuated disease variants have been described, suggesting that a wider clinical spectrum of BRAT1-associated neurodegeneration exists than was previously thought. Here, we report two affected siblings with compound heterozygous truncating mutations in BRAT1 and intra-familial phenotypic heterogeneity, with a less severe disease course in the female sibling. This phenotypic variability should be taken into account when treating patients with BRAT1-associated neurodegenerative disease. Mildly affected individuals with BRAT1 mutations show that BRAT1 must be considered as a cause in childhood refractory epilepsy and microcephaly with survival beyond infancy. © 2016 Wiley Periodicals, Inc.

Keywords: BRAT1; microcephaly; multifocal seizures; neurodegeneration.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Age of Onset
  • Alleles
  • Exome
  • Fatal Outcome
  • Female
  • Genes, Recessive
  • Genetic Association Studies*
  • Genetic Loci
  • Genotype
  • High-Throughput Nucleotide Sequencing
  • Humans
  • Infant
  • Male
  • Mutation*
  • Neurodegenerative Diseases / diagnosis*
  • Neurodegenerative Diseases / genetics*
  • Nuclear Proteins / genetics*
  • Pedigree
  • Phenotype*
  • Siblings

Substances

  • BRAT1 protein, human
  • Nuclear Proteins