The need for genetic study to diagnose some cases of distal renal tubular acidosis
Nefrologia. 2016 Sep-Oct;36(5):552-555.
doi: 10.1016/j.nefro.2016.06.008.
Epub 2016 Aug 1.
[Article in
English,
Spanish]
Affiliations
- 1 Servicio de Nefrología, Hospital General de Segovia, Segovia, España. Electronic address: [email protected].
- 2 Grupo de Genética y Biología del Desarrollo de las Enfermedades Renales, Laboratorio de Nefrología (n.° 11), Instituto de Investigación Sanitaria (IDIS), Complexo Hospitalario de Santiago de Compostela (CHUS), Santiago de Compostela, España.
- 3 Servicio de Nefrología, Hospital General de Segovia, Segovia, España.
Abstract
We describe the case of a young woman who was diagnosed with advanced kidney disease, with an incidental finding of nephrocalcinosis of unknown aetiology, having been found asymptomatic throughout her life. The genetic study by panels of known genes associated with tubulointerstitial disease allowed us to discover autosomal dominant distal renal tubular acidosis associated with a de novo mutation in exon 14 of the SLC4A1 gene, which would have been impossible to diagnose clinically due to the advanced nature of the kidney disease when it was discovered.
Keywords:
Acidosis tubular renal distal autosómica dominante; Autosomal dominant distal renal tubular acidosis; Chronic kidney disease; Insuficiencia renal crónica; NGS Genetic panels; Nefrocalcinosis; Nephrocalcinosis; Paneles genéticos NGS.
Copyright © 2016 Sociedad Española de Nefrología. Published by Elsevier España, S.L.U. All rights reserved.
MeSH terms
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Acidosis, Renal Tubular / diagnosis*
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Acidosis, Renal Tubular / genetics*
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Adult
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Anion Exchange Protein 1, Erythrocyte / genetics*
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Exons
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Female
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Humans
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Kidney / physiopathology
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Mutation
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Nephrocalcinosis / etiology
Substances
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Anion Exchange Protein 1, Erythrocyte
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SLC4A1 protein, human