miR-322/-503 cluster is expressed in the earliest cardiac progenitor cells and drives cardiomyocyte specification

Proc Natl Acad Sci U S A. 2016 Aug 23;113(34):9551-6. doi: 10.1073/pnas.1608256113. Epub 2016 Aug 10.

Abstract

Understanding the mechanisms of early cardiac fate determination may lead to better approaches in promoting heart regeneration. We used a mesoderm posterior 1 (Mesp1)-Cre/Rosa26-EYFP reporter system to identify microRNAs (miRNAs) enriched in early cardiac progenitor cells. Most of these miRNA genes bear MESP1-binding sites and active histone signatures. In a calcium transient-based screening assay, we identified miRNAs that may promote the cardiomyocyte program. An X-chromosome miRNA cluster, miR-322/-503, is the most enriched in the Mesp1 lineage and is the most potent in the screening assay. It is specifically expressed in the looping heart. Ectopic miR-322/-503 mimicking the endogenous temporal patterns specifically drives a cardiomyocyte program while inhibiting neural lineages, likely by targeting the RNA-binding protein CUG-binding protein Elav-like family member 1 (Celf1). Thus, early miRNAs in lineage-committed cells may play powerful roles in cell-fate determination by cross-suppressing other lineages. miRNAs identified in this study, especially miR-322/-503, are potent regulators of early cardiac fate.

Keywords: cardiomyocyte; miR-322; miR-424; miR-503; microRNA.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Bacterial Proteins / genetics
  • Bacterial Proteins / metabolism
  • Basic Helix-Loop-Helix Transcription Factors / genetics*
  • Basic Helix-Loop-Helix Transcription Factors / metabolism
  • CELF1 Protein / genetics
  • CELF1 Protein / metabolism
  • Cell Differentiation
  • Cell Lineage / genetics
  • Embryo, Mammalian
  • Gene Expression Profiling
  • Gene Expression Regulation, Developmental*
  • Genes, Reporter
  • Integrases / genetics
  • Integrases / metabolism
  • Luminescent Proteins / genetics
  • Luminescent Proteins / metabolism
  • Mesoderm / cytology
  • Mesoderm / growth & development
  • Mesoderm / metabolism
  • Mice
  • Mice, Transgenic
  • MicroRNAs / genetics*
  • MicroRNAs / metabolism
  • Morphogenesis / genetics
  • Mouse Embryonic Stem Cells / cytology
  • Mouse Embryonic Stem Cells / metabolism*
  • Myocytes, Cardiac / cytology
  • Myocytes, Cardiac / metabolism*
  • Primary Cell Culture
  • RNA, Untranslated / genetics
  • RNA, Untranslated / metabolism
  • Signal Transduction

Substances

  • Bacterial Proteins
  • Basic Helix-Loop-Helix Transcription Factors
  • CELF1 Protein
  • CELF1 protein, mouse
  • Gt(ROSA)26Sor non-coding RNA, mouse
  • Luminescent Proteins
  • MIRN322 microRNA, mouse
  • Mesp1 protein, mouse
  • MicroRNAs
  • Mirn503 microRNA, mouse
  • RNA, Untranslated
  • yellow fluorescent protein, Bacteria
  • Cre recombinase
  • Integrases