CMV70-3P miRNA contributes to the CMV mediated glioma stemness and represents a target for glioma experimental therapy

Oncotarget. 2017 Apr 18;8(16):25989-25999. doi: 10.18632/oncotarget.11175.

Abstract

Glioblastoma multiforme (GBM) is a rapidly progressive brain tumor with a median survival of 15-19 months. Therapeutic resistance and recurrence of the disease is attributed to cancer stem cells (CSC). Here, we report that CMV70-3P miRNA encoded by CMV increases GBM CSC stemness. Inhibition of CMV70-3P expression using oligo inhibitors significantly attenuated the ability of primary glioma cells to proliferate and form neurospheres. At the molecular level, we show that CM70-3P increases expression of cellular SOX2. Collectively, these findings indicate that CMV70-3P is a potential regulator of CMV- mediated glioma progression and cancer stemness.

Keywords: brain tumor; cytomegalovirus; glioma stem cells; miRNA.

MeSH terms

  • AC133 Antigen / metabolism
  • Cell Line, Tumor
  • Cell Movement / genetics
  • Cytomegalovirus / physiology*
  • Cytomegalovirus Infections / complications*
  • Cytomegalovirus Infections / virology*
  • Gene Expression
  • Glioma / drug therapy
  • Glioma / etiology*
  • Glioma / metabolism*
  • Glioma / pathology
  • Humans
  • MicroRNAs*
  • Neoplastic Stem Cells / metabolism*
  • Phenotype
  • Promoter Regions, Genetic
  • RNA, Viral*
  • SOXB1 Transcription Factors / genetics

Substances

  • AC133 Antigen
  • MicroRNAs
  • RNA, Viral
  • SOXB1 Transcription Factors