Stroke induces specific alteration of T memory compartment controlling auto-reactive CNS antigen-specific T cell responses

J Neurol Sci. 2016 Sep 15:368:77-83. doi: 10.1016/j.jns.2016.06.039. Epub 2016 Jun 17.

Abstract

Whether and when auto-reactivity after stroke occurs is still a matter of debate. By using overlapping 15mer peptide pools consisting of myelin basic protein (MBP) and myelin oligodendrocyte glycoprotein (MOG) we show increased frequencies of immunodominant MOG- and MBP T cell responses in acute ischemic stroke which were associated with reduced frequencies of naïve T cells as well as CD8+ TEMRA cells. Auto-reactive CNS antigen-specific T cells responses as well as alterations of T cell subpopulations normalized in long-term follow up after stroke. Our findings suggest that stroke-induced immunodepression might function as an adaptive mechanism in order to inhibit harmful and long-lasting CNS antigen-specific immune responses.

Keywords: Auto-reactive T cell; MBP; MOG; Stroke; T memory cell.

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • Autoimmunity*
  • Brain Ischemia / immunology*
  • Brain Ischemia / therapy
  • Enzyme-Linked Immunospot Assay
  • Female
  • Flow Cytometry
  • Follow-Up Studies
  • Humans
  • Immunologic Memory*
  • Interferon-gamma / metabolism
  • Male
  • Middle Aged
  • Myelin Basic Protein / metabolism
  • Myelin-Oligodendrocyte Glycoprotein / metabolism
  • Neuroimmunomodulation
  • Stroke / immunology*
  • Stroke / therapy
  • T-Lymphocyte Subsets / immunology*

Substances

  • MBP protein, human
  • MOG protein, human
  • Myelin Basic Protein
  • Myelin-Oligodendrocyte Glycoprotein
  • Interferon-gamma