Bioassay-Guided Fractionation and In Vitro Antiproliferative Effects of Fractions of Artemisia nilagirica on THP-1 cell line

Nutr Cancer. 2016 Oct;68(7):1210-24. doi: 10.1080/01635581.2016.1205900. Epub 2016 Aug 11.

Abstract

ABSTACT Artemisia nilagirica (Clarke) is a widely used medicinal herb in Indian traditional system of medicine. Therefore, the present study was designed to evaluate the effects of A. nilagirica extracts/fractions on inhibition of proliferation and apoptosis in a human monocytic leukemia (THP-1) cell line. The crude extracts (A. nilagirica ethyl acetate extract [ANE] and A. nilagirica methanolic extract [ANA]) showed cytotoxic activity toward THP-1 cells with the IC50 values of 38.21 ± 7.37 and 132.41 ± 7.19 µg/ml, respectively. However, the cytotoxic activity of active fractions (ANE-B and ANM-9) obtained after column chromatography was found to be much more pronounced than their parent extracts. The IC50 values of ANE-B and ANM-9 were found to be 27.04 ± 2.54 µg/ml and 12.70 ± 4.79 µg/ml, respectively, suggesting greater susceptibility of the malignant cells. Cell cycle analysis and terminal deoxynucleotidyl transferase (TdT)-mediated dUTP nick-end-labeling (TUNEL) assay revealed that inhibition of cell growth by A. nilagirica fractions on THP-1 cells was mediated by apoptosis. Active fractions of A. nilagirica increased the expression levels of caspase-3, -7, and poly-ADP-ribose polymerase (PARP), a critical member of the apoptotic pathway. These results suggested that active fractions of A. nilagirica may play a promising role in growth suppression by inducing apoptosis in human monocytic leukemic cells via mitochondria-dependent and death receptor-dependent apoptotic pathways.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Anticarcinogenic Agents / adverse effects
  • Anticarcinogenic Agents / chemistry
  • Anticarcinogenic Agents / isolation & purification*
  • Anticarcinogenic Agents / pharmacology
  • Antineoplastic Agents, Phytogenic / adverse effects
  • Antineoplastic Agents, Phytogenic / chemistry
  • Antineoplastic Agents, Phytogenic / isolation & purification*
  • Antineoplastic Agents, Phytogenic / pharmacology
  • Apoptosis / drug effects*
  • Artemisia / chemistry*
  • Biological Assay
  • Caspase 3 / chemistry
  • Caspase 3 / genetics
  • Caspase 3 / metabolism
  • Caspase 7 / chemistry
  • Caspase 7 / genetics
  • Caspase 7 / metabolism
  • Cell Proliferation / drug effects
  • Cells, Cultured
  • Female
  • Gene Expression Regulation, Neoplastic / drug effects
  • Humans
  • India
  • Inhibitory Concentration 50
  • Leukemia, Monocytic, Acute / drug therapy*
  • Leukemia, Monocytic, Acute / metabolism
  • Leukemia, Monocytic, Acute / pathology
  • Macrophages, Peritoneal / cytology
  • Macrophages, Peritoneal / drug effects*
  • Mice, Inbred BALB C
  • Neoplasm Proteins / agonists
  • Neoplasm Proteins / antagonists & inhibitors
  • Neoplasm Proteins / genetics
  • Neoplasm Proteins / metabolism
  • Plant Extracts / adverse effects
  • Plant Extracts / chemistry
  • Plant Extracts / isolation & purification
  • Plant Extracts / pharmacology
  • Poly(ADP-ribose) Polymerases / chemistry
  • Poly(ADP-ribose) Polymerases / genetics
  • Poly(ADP-ribose) Polymerases / metabolism
  • THP-1 Cells

Substances

  • Anticarcinogenic Agents
  • Antineoplastic Agents, Phytogenic
  • Neoplasm Proteins
  • Plant Extracts
  • Poly(ADP-ribose) Polymerases
  • Caspase 3
  • Caspase 7