BDNF mRNA abundance regulated by antidromic action potentials and AP-LTD in hippocampus

Neurosci Lett. 2016 Dec 2:635:97-102. doi: 10.1016/j.neulet.2016.10.023. Epub 2016 Oct 17.

Abstract

Action-potential-induced LTD (AP-LTD) is a form of synaptic plasticity that reduces synaptic strength in CA1 hippocampal neurons firing antidromically during sharp-wave ripples. This firing occurs during slow-wave sleep and quiet moments of wakefulness, which are periods of offline replay of neural sequences learned during encoding sensory information. Here we report that rapid and persistent down-regulation of different mRNA transcripts of the BDNF gene accompanies AP-LTD, and that AP-LTD is abolished in mice with the BDNF gene knocked out in CA1 hippocampal neurons. These findings increase understanding of the mechanism of AP-LTD and the cellular mechanisms of memory consolidation.

Keywords: Antidromic action potentials; BDNF; LTD; Sharp wave ripple complexes; Slow wave sleep.

MeSH terms

  • Action Potentials*
  • Animals
  • Brain-Derived Neurotrophic Factor / genetics
  • Brain-Derived Neurotrophic Factor / metabolism*
  • Calcium Channels, L-Type / metabolism
  • Hippocampus / physiology*
  • Long-Term Synaptic Depression*
  • Male
  • RNA, Messenger / metabolism*
  • Rats, Sprague-Dawley

Substances

  • Brain-Derived Neurotrophic Factor
  • Calcium Channels, L-Type
  • RNA, Messenger