Co-regulation of Cxcl1 and versican in the inflammatory response to UVB induced reactive oxygen species in skin photo-tumorigenesis

J Dermatol Sci. 2017 Feb;85(2):140-143. doi: 10.1016/j.jdermsci.2016.10.011. Epub 2016 Oct 20.
No abstract available

Publication types

  • Letter

MeSH terms

  • Animals
  • Carcinogenesis / genetics
  • Carcinogenesis / metabolism*
  • Carcinogenesis / radiation effects
  • Chemokine CXCL1 / genetics
  • Chemokine CXCL1 / metabolism*
  • DNA Glycosylases / genetics
  • Fibroblasts / radiation effects
  • Gene Expression Profiling
  • Gene Expression Regulation, Neoplastic*
  • Gene Knockdown Techniques
  • Inflammation / metabolism
  • Mice
  • Mice, Knockout
  • Neoplasms, Radiation-Induced / genetics
  • Neoplasms, Radiation-Induced / metabolism*
  • Neoplasms, Radiation-Induced / pathology
  • RNA Interference
  • RNA, Small Interfering / metabolism
  • Reactive Oxygen Species / metabolism
  • Signal Transduction
  • Skin / metabolism
  • Skin / pathology
  • Skin / radiation effects
  • Skin Neoplasms / genetics
  • Skin Neoplasms / metabolism*
  • Skin Neoplasms / pathology
  • Ultraviolet Rays / adverse effects
  • Up-Regulation
  • Versicans / genetics
  • Versicans / metabolism*

Substances

  • Chemokine CXCL1
  • Cxcl1 protein, mouse
  • RNA, Small Interfering
  • Reactive Oxygen Species
  • Vcan protein, mouse
  • Versicans
  • DNA Glycosylases
  • Ogg1 protein, mouse