The ethyl acetate fraction of corn silk exhibits dual antioxidant and anti-glycation activities and protects insulin-secreting cells from glucotoxicity

BMC Complement Altern Med. 2016 Nov 3;16(1):432. doi: 10.1186/s12906-016-1382-8.

Abstract

Background: In this study, we aimed to develop a Stigmata Maydis (corn silk) fraction with dual bio-activities against oxidative stress and protein glycation to protect β-cells from diabetes-induced failure.

Methods: Corn silk fractions were prepared by partition and chemically characterised by thin-layer chromatography. Free radical scavenging assay, glycation assay, and cell-based viability test (neutral red) were employed to decide the best fraction. Cell death analysis was executed by annexin V/ Propidium iodide staining. Cell proliferation was measured by WST-1. Finally, β-cell function was evaluated by β-cell marker gene expression (RT-PCR) and acute insulin secretion test.

Results: Four corn silk fractions were prepared from an ethanolic crude extract of corn silk. In vitro assays indicate ethyl acetate fraction (YMS-EA) was the most potent fraction. YMS-EA also attenuated the hydrogen peroxide- or methylglyoxal-induced induction of reactive oxygen species, reduction of cell viability, and inhibition of cell proliferation. However, YMS-EA was unable to prevent hydrogen peroxide-induced apoptosis or advanced glycation end-products-induced toxicity. Under hyperglycemic conditions, YMS-EA effectively reduced ROS levels, improved mRNA expression of insulin, glucokinase, and PDX-1, and enhanced glucose-stimulated insulin secretion. The similarity of bioactivities among apigenin, luteolin, and YMS-EA indicated that dual activities of YMS-EA might be derived from those compounds.

Conclusions: We concluded that YMS-EA fraction could be developed as a preventive food agent against the glucotoxicity to β-cells in Type 2 diabetes.

Keywords: Advanced glycation end products; Glucotoxicity; Methylglyoxal; Reactive oxygen species; Stigmata Maydis (corn silk); β-cell failure.

MeSH terms

  • Acetates / chemistry
  • Animals
  • Antioxidants / chemistry
  • Antioxidants / pharmacology*
  • Cell Line
  • Cell Survival / drug effects
  • Female
  • Glycation End Products, Advanced / analysis
  • Glycation End Products, Advanced / metabolism
  • Hydrogen Peroxide / toxicity
  • Male
  • Mice
  • Oxidative Stress / drug effects*
  • Plant Extracts / chemistry
  • Plant Extracts / pharmacology*
  • Rats
  • Reactive Oxygen Species / analysis
  • Reactive Oxygen Species / metabolism
  • Zea mays / chemistry*

Substances

  • Acetates
  • Antioxidants
  • Glycation End Products, Advanced
  • Plant Extracts
  • Reactive Oxygen Species
  • ethyl acetate
  • Hydrogen Peroxide