Correlation between the high expression levels of cancer-germline genes with clinical characteristics in esophageal squamous cell carcinoma

Histol Histopathol. 2017 Aug;32(8):793-803. doi: 10.14670/HH-11-847. Epub 2016 Nov 21.

Abstract

Antigens encoded by cancer-germline genes are attractive targets for cancer immunotherapy. In this study, we aimed to evaluate the mRNA expression of cancer-germline genes, expression of the encoded proteins in patients with esophageal squamous cell carcinoma (ESCC) and their correlations with clinical characteristics. In addition, the effects of downregulation cancer-germline genes on ESCC cells were assessed in vitro. Our results showed that cancer-germline genes were frequently expressed in ESCC samples. The positive rates of in ESCC samples were: 87% of MAGE-A3, 60% of MAGE-A4, 65% of MAGE-C2, and 20% of NY-ESO-1 at mRNA level. MAGE-A3 expression was associated with age, lymph node metastasis and tumor stage (all P<0.05), while MAGE-C2 expression was only associated with tumor stage (P<0.05). Furthermore, the MAGE-A3 expressing patients had a poorer overall survival (P<0.05). Multivariate analysis identified MAGE-A3 as an independent poor prognostic marker in ESCC. In vitro assay, ESCC cell lines treated with specific siRNAs to down-regulate MAGE-A3 and MAGE-C2 resulted in decreased colony-formation and migration ability (P<0.05). Epithelial marker E-cadherin was up-regulated in siRNA-MAGE-A3/C2 cells compared to controls, whereas mesenchymal markers Vimentin, N-cadherin and Slug were downregulated (all P<0.05), suggesting a role for MAGE-A3/C2 in ESCC metastasis through inducing epithelial-mesenchymal transition. The present study revealed that cancer-germline genes and their encoded proteins were frequently expressed in ESCC tumor samples and were related to poor prognosis. Thus, cancer-germline genes may serve as useful biomarkers and potential targets for ESCC patients.

MeSH terms

  • Aged
  • Antigens, Neoplasm / genetics*
  • Apoptosis
  • Biomarkers, Tumor / genetics
  • Cadherins / genetics
  • Carcinoma, Squamous Cell / genetics*
  • Cell Line, Tumor
  • Down-Regulation
  • Epithelial-Mesenchymal Transition
  • Esophageal Neoplasms / genetics*
  • Female
  • Gene Expression Regulation, Neoplastic*
  • Humans
  • Immunotherapy
  • Male
  • Membrane Proteins / genetics
  • Middle Aged
  • Multivariate Analysis
  • Neoplasm Proteins / genetics
  • Prognosis
  • RNA, Messenger / metabolism
  • RNA, Small Interfering / metabolism
  • Snail Family Transcription Factors / genetics
  • Treatment Outcome
  • Vimentin / genetics

Substances

  • Antigens, Neoplasm
  • Biomarkers, Tumor
  • CTAG1B protein, human
  • Cadherins
  • MAGEA3 protein, human
  • MAGEA4 protein, human
  • MAGEC2 protein, human
  • Membrane Proteins
  • Neoplasm Proteins
  • RNA, Messenger
  • RNA, Small Interfering
  • SNAI1 protein, human
  • Snail Family Transcription Factors
  • Vimentin