Design, synthesis, and biological evaluation of novel 1-oxo-1,2,3,4-tetrahydropyrazino[1,2-a]indole-3-carboxamide analogs in MCF-7 and MDA-MB-468 breast cancer cell lines

Bioorg Med Chem Lett. 2017 Feb 1;27(3):607-611. doi: 10.1016/j.bmcl.2016.12.006. Epub 2016 Dec 5.

Abstract

A series of novel 1-oxo-1,2,3,4-tetrahydropyrazino[1,2-a]indole-3-carboxamide analogs were designed and synthesized for developing pyrazinoindolone scaffolds as anti-breast cancer agents. Compounds 1h and 1i, having a furan-2-yl-methylamide and benzylamide group, respectively, exhibited more potent cytotoxicity in MDA-MB-468 triple-negative breast cancer (TNBC) cells than compounds possessing aliphatic groups. Compounds 2a and 2b, as (R)-enantiomers of 1h and 1i, also had inhibitory activity against MDA-MB-468 cells. Moreover, analogs (3a-b and 3d-e) bearing a benzyl group at the N-2 position showed more potent activity than gefitinib, as a potent EFGR-TK inhibitor. Especially, compound 3a exhibited selective cytotoxic activity against MDA-MB-468 cells; it also had a synergistic effect in combination with gefitinib against MDA-MB-468 cells. In addition, we confirmed that compounds 3a and 3d inhibit phosphorylation of Akt in MDA-MB-468 cells using western blot analysis. Therefore, these 1-oxo-1,2,3,4-tetrahydropyrazino[1,2-a]indole-3-carboxamide analogs may be helpful for investigating new anti-TNBC agents.

Keywords: Anti-breast cancer activity; Gefitinib-resistant TNBC; Inhibition of p-Akt; Pyrazinoindolone scaffold.

MeSH terms

  • Amides / chemical synthesis
  • Amides / chemistry*
  • Amides / pharmacology*
  • Antineoplastic Agents / chemical synthesis*
  • Antineoplastic Agents / chemistry
  • Antineoplastic Agents / pharmacology
  • Breast Neoplasms / metabolism
  • Breast Neoplasms / pathology
  • Cell Line, Tumor
  • Cell Survival / drug effects*
  • Drug Design*
  • Drug Resistance, Neoplasm / drug effects
  • Female
  • Gefitinib
  • Humans
  • Indoles / chemistry*
  • Indoles / pharmacology
  • MCF-7 Cells
  • Phosphorylation / drug effects
  • Proto-Oncogene Proteins c-akt / metabolism
  • Quinazolines / pharmacology
  • Structure-Activity Relationship

Substances

  • Amides
  • Antineoplastic Agents
  • Indoles
  • Quinazolines
  • Proto-Oncogene Proteins c-akt
  • Gefitinib