Reduction of hyperthermic ischemic acidosis by a conditioning event in cats

Stroke. 1989 Oct;20(10):1357-60. doi: 10.1161/01.str.20.10.1357.

Abstract

We investigated the effects of multiple episodes of cerebral ischemia on intracellular brain pH using in vivo phosphorus-31 nuclear magnetic resonance spectroscopy. Four cats were subjected to two 16-minute episodes of complete global cerebral ischemia 6 hours apart; the second episode occurred under hyperthermic conditions (mean +/- SD body temperature 40.8 +/- 0.4 degrees C). Intracellular pH in these four cats was compared with that in nine cats subjected to a single 16-minute episode of complete global cerebral ischemia under hyperthermic conditions (mean +/- SD body temperature 40.6 +/- 0.2 degrees C). Intracellular pH during hyperthermic recirculation was significantly (p less than 0.03) greater in cats subjected to a previous ischemic event than that in cats subjected to only a single hyperthermic ischemic event. We speculate that the induction of heat shock proteins by an initial ischemic event may protect brain tissue from further ischemic insult.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Acidosis / metabolism*
  • Animals
  • Brain / metabolism*
  • Brain Ischemia / metabolism*
  • Cats
  • Heat-Shock Proteins / metabolism*
  • Hydrogen-Ion Concentration
  • Hyperthermia, Induced*
  • Magnetic Resonance Spectroscopy

Substances

  • Heat-Shock Proteins