Enteroviruses infect human enteroids and induce antiviral signaling in a cell lineage-specific manner

Proc Natl Acad Sci U S A. 2017 Feb 14;114(7):1672-1677. doi: 10.1073/pnas.1617363114. Epub 2017 Jan 30.

Abstract

Enteroviruses are among the most common viral infectious agents of humans and are primarily transmitted by the fecal-oral route. However, the events associated with enterovirus infections of the human gastrointestinal tract remain largely unknown. Here, we used stem cell-derived enteroids from human small intestines to study enterovirus infections of the intestinal epithelium. We found that enteroids were susceptible to infection by diverse enteroviruses, including echovirus 11 (E11), coxsackievirus B (CVB), and enterovirus 71 (EV71), and that contrary to an immortalized intestinal cell line, enteroids induced antiviral and inflammatory signaling pathways in response to infection in a virus-specific manner. Furthermore, using the Notch inhibitor dibenzazepine (DBZ) to drive cellular differentiation into secretory cell lineages, we show that although goblet cells resist E11 infection, enteroendocrine cells are permissive, suggesting that enteroviruses infect specific cell populations in the human intestine. Taken together, our studies provide insights into enterovirus infections of the human intestine, which could lead to the identification of novel therapeutic targets and/or strategies to prevent or treat infections by these highly clinically relevant viruses.

Keywords: enteroendocrine cells; enteroid; enterovirus; goblet cells; innate immune.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Caco-2 Cells
  • Cell Lineage / drug effects
  • Cell Lineage / genetics
  • Cells, Cultured
  • Dibenzazepines / pharmacology
  • Disease Resistance / genetics
  • Enterovirus / physiology*
  • Enterovirus Infections / metabolism
  • Enterovirus Infections / virology*
  • Enzyme Inhibitors / pharmacology
  • Gene Expression Profiling / methods
  • Host-Pathogen Interactions
  • Humans
  • Intestinal Mucosa / cytology
  • Intestinal Mucosa / metabolism
  • Intestinal Mucosa / virology
  • Intestine, Small / cytology
  • Intestine, Small / metabolism
  • Intestine, Small / virology*
  • Organoids / cytology
  • Organoids / metabolism
  • Organoids / virology*
  • Signal Transduction / genetics

Substances

  • Dibenzazepines
  • Enzyme Inhibitors
  • dibenzazepine