Tissue-specific differentiation of colonic macrophages requires TGFβ receptor-mediated signaling

Mucosal Immunol. 2017 Nov;10(6):1387-1399. doi: 10.1038/mi.2016.142. Epub 2017 Feb 1.

Abstract

Intestinal macrophages (mφ) form one of the largest populations of mφ in the body and are vital for the maintenance of gut homeostasis. They have several unique properties and are derived from local differentiation of classical Ly6Chi monocytes, but the factors driving this tissue-specific process are not understood. Here we have used global transcriptomic analysis to identify a unique homeostatic signature of mature colonic mφ that is acquired as they differentiate in the mucosa. By comparing the analogous monocyte differentiation process found in the dermis, we identify TGFβ as an indispensable part of monocyte differentiation in the intestine and show that it enables mφ to adapt precisely to the requirements of their environment. Importantly, TGFβR signaling on mφ has a crucial role in regulating the accumulation of monocytes in the mucosa, via mechanisms that are distinct from those used by IL10.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antigens, Ly / metabolism
  • Cell Differentiation
  • Cells, Cultured
  • Cellular Microenvironment
  • Colon / immunology*
  • Female
  • Gene Expression Profiling
  • Homeostasis
  • Macrophages / immunology*
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Monocytes / immunology*
  • Organ Specificity
  • Receptors, Transforming Growth Factor beta / genetics
  • Receptors, Transforming Growth Factor beta / metabolism*
  • Signal Transduction
  • Transcriptome
  • Transforming Growth Factor beta / metabolism*

Substances

  • Antigens, Ly
  • Ly-6C antigen, mouse
  • Receptors, Transforming Growth Factor beta
  • Transforming Growth Factor beta