Platelet adhesion to subendothelial structures under flow conditions: no effect of the lipoxygenase product 13-HODE

Thromb Haemost. 1989 Sep 29;62(2):802-6.

Abstract

It has been shown that endothelial cells can convert linoleic acid to 13-hydroxyoctadecadienoic acid (13-HODE) and it has been suggested that 13-HODE has non-thrombogenic properties. However, no direct evidence has been presented that indicates that 13-HODE indeed modulates platelet-vessel wall interaction. In this study we have bound a purified 13-HODE to a thrombogenic surface and its effect on platelet adhesion was studied and compared to the effects of an analogous hydroxy fatty acid, 15-hydroxyeicosatetraenoic acid (15-HETE). The effect of 13-HODE on platelet adhesion was studied both under static and flow conditions. In this report we show that binding of up to 40 times the physiological concentration to a thrombogenic surface has no inhibitory effect on platelet adhesion under static or flow conditions. We conclude that 13-HODE is not an important regulatory substance in platelet-subendothelium interaction, although this does not exclude it has a putative anti-adhesive role on intact endothelium.

MeSH terms

  • Cells, Cultured
  • Chromatography, High Pressure Liquid
  • Endothelium, Vascular / drug effects*
  • Endothelium, Vascular / metabolism
  • Humans
  • Hydroxyeicosatetraenoic Acids / pharmacology
  • Linoleic Acids / metabolism
  • Linoleic Acids / pharmacology*
  • Perfusion
  • Platelet Adhesiveness / drug effects*

Substances

  • Hydroxyeicosatetraenoic Acids
  • Linoleic Acids
  • 13-hydroxy-9,11-octadecadienoic acid
  • 15-hydroxy-5,8,11,13-eicosatetraenoic acid