Gene expression analysis of hypersensitivity to mosquito bite, chronic active EBV infection and NK/T-lymphoma/leukemia

Leuk Lymphoma. 2017 Nov;58(11):2683-2694. doi: 10.1080/10428194.2017.1304762. Epub 2017 Apr 3.

Abstract

The human herpes virus, Epstein-Barr virus (EBV), is a known oncogenic virus and plays important roles in life-threatening T/NK-cell lymphoproliferative disorders (T/NK-cell LPD) such as hypersensitivity to mosquito bite (HMB), chronic active EBV infection (CAEBV), and NK/T-cell lymphoma/leukemia. During the clinical courses of HMB and CAEBV, patients frequently develop malignant lymphomas and the diseases passively progress sequentially. In the present study, gene expression of CD16(-)CD56(+)-, EBV(+) HMB, CAEBV, NK-lymphoma, and NK-leukemia cell lines, which were established from patients, was analyzed using oligonucleotide microarrays and compared to that of CD56brightCD16dim/- NK cells from healthy donors. Principal components analysis showed that CAEBV and NK-lymphoma cells were relatively closely located, indicating that they had similar expression profiles. Unsupervised hierarchal clustering analyses of microarray data and gene ontology analysis revealed specific gene clusters and identified several candidate genes responsible for disease that can be used to discriminate each category of NK-LPD and NK-cell lymphoma/leukemia.

Keywords: CAEBV; HMB; NK-LPD; NK-lymphoma/leukemia; microarray.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Aged
  • Animals
  • Cell Line, Tumor
  • Child
  • Chronic Disease
  • Cluster Analysis
  • Culicidae / immunology
  • Epstein-Barr Virus Infections / complications
  • Epstein-Barr Virus Infections / genetics*
  • Female
  • Gene Expression Profiling*
  • Gene Ontology
  • Gene Regulatory Networks / genetics
  • Humans
  • Hypersensitivity / genetics*
  • Hypersensitivity / immunology
  • Insect Bites and Stings / immunology
  • Killer Cells, Natural / metabolism
  • Killer Cells, Natural / pathology
  • Leukemia / complications
  • Leukemia / genetics*
  • Leukemia / pathology
  • Lymphoma, T-Cell / complications
  • Lymphoma, T-Cell / genetics*
  • Lymphoma, T-Cell / pathology
  • Male
  • Middle Aged
  • Principal Component Analysis
  • Signal Transduction / genetics
  • Young Adult