Relaxin and Matrix Metalloproteinase-9 in Angiotensin II-Induced Abdominal Aortic Aneurysms

Circ J. 2017 May 25;81(6):888-890. doi: 10.1253/circj.CJ-17-0229. Epub 2017 Apr 18.

Abstract

Background: This study determined whether relaxin or matrix metalloproteinase (MMP)-9 influences angiotensin II (AngII)-induced abdominal aortic aneurysms (AAA).

Methods and results: Male C57BL/6 or apolipoprotein E-/-mice were infused with AngII with or without relaxin. Relaxin did not influence AngII-induced AAA in either mouse strain. Infusion of AngII reduced, but relaxin increased, MMP-9 mRNA in macrophages. We then determined the effects of MMP-9 deficiency on AAA in apolipoprotein E-/-mice. MMP-9 deficiency led to AAA formation in the absence of AngII, and augmented AngII-induced aortic rupture and AAA incidence.

Conclusions: MMP-9 deficiency augmented AngII-induced AAA.

Keywords: Abdominal aortic aneurysm; Matrix metalloproteinase; Relaxin.

MeSH terms

  • Angiotensin II / adverse effects*
  • Angiotensin II / pharmacology
  • Animals
  • Aortic Aneurysm, Abdominal / chemically induced
  • Aortic Aneurysm, Abdominal / genetics
  • Aortic Aneurysm, Abdominal / metabolism*
  • Aortic Aneurysm, Abdominal / pathology
  • Apolipoproteins E / deficiency
  • Matrix Metalloproteinase 9 / genetics
  • Matrix Metalloproteinase 9 / metabolism*
  • Mice
  • Mice, Knockout
  • Relaxin / biosynthesis*
  • Relaxin / genetics

Substances

  • Apolipoproteins E
  • Angiotensin II
  • Relaxin
  • Matrix Metalloproteinase 9
  • Mmp9 protein, mouse