MicroRNA-212 inhibits the metastasis of nasopharyngeal carcinoma by targeting SOX4

Oncol Rep. 2017 Jul;38(1):82-88. doi: 10.3892/or.2017.5641. Epub 2017 May 15.

Abstract

MicroRNAs are important factors in the pathogenic processes of human types of cancers including nasopharyngeal carcinoma (NPC). In the present study, we confirmed that the microRNA-212 expression level was significantly decreased both in NPC tissues and NPC cell lines. Decreased expression of miR-212 was associated with advanced tumor-node-metastasis (TNM) stage and metastasis of NPC. Patients with a lower level of miR-212 had significantly decreased rates of overall and disease-free survival. Functional experiments showed that forced expression of miR-212 inhibited the migration and invasion of NPC cells while inhibition of miR-212 increased the migration and invasion of NPC cells. Furthermore, the results of luciferase assay, qRT-PCR and western blotting showed that SOX4 was the direct downstream target of miR-212 in NPC cells. In addition, we further confirmed that miR-212 exerted its inhibitory influence on the migration and invasion of NPC cells by targeting SOX4.

MeSH terms

  • Apoptosis
  • Carcinoma / genetics
  • Carcinoma / prevention & control*
  • Carcinoma / secondary
  • Cell Movement*
  • Cell Proliferation
  • Female
  • Gene Expression Regulation, Neoplastic*
  • Humans
  • Lymphatic Metastasis
  • Male
  • MicroRNAs / genetics*
  • Middle Aged
  • Nasopharyngeal Carcinoma
  • Nasopharyngeal Neoplasms / genetics
  • Nasopharyngeal Neoplasms / prevention & control*
  • Nasopharyngeal Neoplasms / secondary
  • Neoplasm Invasiveness
  • Prognosis
  • SOXC Transcription Factors / antagonists & inhibitors*
  • Tumor Cells, Cultured

Substances

  • MIRN212 microRNA, human
  • MicroRNAs
  • SOX4 protein, human
  • SOXC Transcription Factors