The behavioral deficits and cognitive impairment are correlated with decreased IGF-II and ERK in depressed mice induced by chronic unpredictable stress

Int J Neurosci. 2017 Dec;127(12):1096-1103. doi: 10.1080/00207454.2017.1337014. Epub 2017 Jun 18.

Abstract

Aim: To investigate the behavioral deficits, cognitive impairment and possible mechanisms induced by chronic unpredictable stress (CUS) in mice of different ages.

Methods: The behaviors and cognition were tested using the open field test, tail suspension test and the Morris water maze. The changes in the insulin-like growth factor II (IGF-II) and the extracellular signal-regulated kinase (ERK) and phosphorylation (p-ERK) in the hippocampus (HP) and prefrontal cortex (PFC) were evaluated by immunohistochemistry.

Results: The 15-month-old mice revealed a significant decline in spontaneous behavior and the learning-memory function and showed a decrease in IGF-II and p-ERK expression levels in HP and PFC. Four weeks of CUS exposure induced significant anxiety- and depression-like behavior and learning-memory function impairment in 3- to 15-month-old mice, and reduced IGF-II and p-ERK expression levels in HP and PFC, compared with control group mice, respectively.

Conclusions: The behavioral deficits and cognitive impairment induced by CUS and aging in mice could be associated with the down-regulated expression of IGF-II and p-ERK in HP and PFC. This role seems to be dependent on the intracellular ERK pathway.

Keywords: CUS; ERK; IGF-II; behavior; learning and memory.

MeSH terms

  • Aging / metabolism
  • Aging / physiology*
  • Animals
  • Anxiety / etiology
  • Anxiety / metabolism
  • Anxiety / physiopathology
  • Behavior, Animal / physiology*
  • Cognitive Dysfunction* / etiology
  • Cognitive Dysfunction* / metabolism
  • Cognitive Dysfunction* / physiopathology
  • Depression* / etiology
  • Depression* / metabolism
  • Depression* / physiopathology
  • Disease Models, Animal
  • Down-Regulation
  • Extracellular Signal-Regulated MAP Kinases / metabolism*
  • Female
  • Hippocampus / metabolism*
  • Insulin-Like Growth Factor II / metabolism*
  • Male
  • Mice
  • Prefrontal Cortex / metabolism*
  • Spatial Learning / physiology
  • Stress, Psychological* / complications
  • Stress, Psychological* / metabolism
  • Stress, Psychological* / physiopathology

Substances

  • IGF2 protein, mouse
  • Insulin-Like Growth Factor II
  • Extracellular Signal-Regulated MAP Kinases