NG2 plays a role in neuroinflammation but is not expressed by immune cells

Acta Neuropathol. 2017 Aug;134(2):325-327. doi: 10.1007/s00401-017-1735-5. Epub 2017 May 31.
No abstract available

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antigens / genetics
  • Antigens, CD / metabolism
  • Bacterial Proteins / genetics
  • Bacterial Proteins / metabolism
  • Central Nervous System / metabolism
  • Central Nervous System / pathology
  • Disease Models, Animal
  • Encephalomyelitis, Autoimmune, Experimental* / genetics
  • Encephalomyelitis, Autoimmune, Experimental* / immunology
  • Encephalomyelitis, Autoimmune, Experimental* / pathology
  • Luminescent Proteins / genetics
  • Luminescent Proteins / metabolism
  • Mice
  • Mice, Transgenic
  • Myelin-Oligodendrocyte Glycoprotein / immunology
  • Myelin-Oligodendrocyte Glycoprotein / toxicity
  • Peptide Fragments / immunology
  • Peptide Fragments / toxicity
  • Pertussis Toxin / toxicity
  • Proteoglycans / deficiency*
  • Proteoglycans / genetics
  • Receptor, Platelet-Derived Growth Factor beta / metabolism
  • Th1 Cells / pathology*
  • Th17 Cells / pathology*

Substances

  • Antigens
  • Antigens, CD
  • Bacterial Proteins
  • Luminescent Proteins
  • Myelin-Oligodendrocyte Glycoprotein
  • Peptide Fragments
  • Proteoglycans
  • chondroitin sulfate proteoglycan 4
  • myelin oligodendrocyte glycoprotein (35-55)
  • yellow fluorescent protein, Bacteria
  • Pertussis Toxin
  • Receptor, Platelet-Derived Growth Factor beta