Role of PET to evaluate coronary microvascular dysfunction in non-ischemic cardiomyopathies

Heart Fail Rev. 2017 Jul;22(4):455-464. doi: 10.1007/s10741-017-9628-1.

Abstract

Coronary microvascular dysfunction (CMD) can result from structural and functional abnormalities at the intramural and small coronary vessel level affecting coronary blood flow autoregulation and consequently leading to impaired coronary flow reserve. CMD often co-exists with epicardial coronary artery disease but is also commonly seen in patients with various forms of heart disease, including dilated, hypertrophic, and infiltrative cardiomyopathies. CMD can go unnoticed without any symptoms, or manifest as angina, and/or dyspnea, and contribute to the development of heart failure, and even sudden death especially when co-existing with myocardial fibrosis. However, whether CMD in non-ischemic cardiomyopathy is a cause or an effect of the underlying cardiomyopathic process, or whether it can be potentially modifiable with specific therapies, remains incompletely understood.

Keywords: Cardiac PET; Coronary flow reserve; Coronary microvascular dysfunction; Non-ischemic cardiomyopathy.

Publication types

  • Review
  • Research Support, N.I.H., Extramural

MeSH terms

  • Blood Flow Velocity
  • Cardiomyopathies / diagnostic imaging*
  • Cardiomyopathies / physiopathology
  • Fibrosis
  • Fractional Flow Reserve, Myocardial
  • Heart Failure / diagnostic imaging*
  • Heart Failure / etiology
  • Heart Failure / physiopathology
  • Humans
  • Image Interpretation, Computer-Assisted*
  • Myocardial Perfusion Imaging*
  • Myocardium / pathology
  • Positron-Emission Tomography*