Tissue-specific posttranslational processing of pre-prosomatostatin encoded by a metallothionein-somatostatin fusion gene in transgenic mice

Cell. 1985 May;41(1):211-9. doi: 10.1016/0092-8674(85)90075-3.

Abstract

The somatostatins are neuropeptides of 14 and 28 amino acids that inhibit the release of growth hormone and other hypophyseal and gastrointestinal peptides. These neuropeptides are cleaved posttranslationally from a common precursor, pre-prosomatostatin. We report here the production and processing of pre-prosomatostatin by transgenic mice carrying a metallothionein-somatostatin fusion gene. The most active site of somatostatin production, as determined by hormone concentrations in the tissues, is the anterior pituitary, a tissue that does not normally synthesize somatostatin-like peptides. Anterior pituitary processed pre-prosomatostatin almost exclusively to the two biologically active peptides, somatostatin-14 and somatostatin-28, whereas the liver and kidney synthesized much smaller quantities of predominantly a 6000 dalton somatostatin-like peptide. The growth of the transgenic mice was normal despite high plasma levels of the somatostatin-like peptides. These studies indicate that proteases which cleave prosomatostatin to somatostatin-28 and somatostatin-14 are not specific to tissues that normally express somatostatin.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • DNA, Recombinant
  • Female
  • Kidney / metabolism
  • Liver / metabolism
  • Male
  • Metallothionein / genetics
  • Mice
  • Organ Specificity
  • Pituitary Gland, Anterior / metabolism
  • Protein Precursors / genetics
  • Protein Precursors / metabolism*
  • Protein Processing, Post-Translational*
  • Somatostatin / biosynthesis
  • Somatostatin / blood
  • Somatostatin / genetics
  • Somatostatin / metabolism*
  • Spleen / metabolism

Substances

  • DNA, Recombinant
  • Protein Precursors
  • Somatostatin
  • Metallothionein