Evidence against VIP as the inhibitory transmitter in non-adrenergic, non-cholinergic nerves supplying the longitudinal muscle of the mouse colon

Br J Pharmacol. 1986 Nov;89(3):599-602. doi: 10.1111/j.1476-5381.1986.tb11161.x.

Abstract

Vasoactive intestinal peptide (VIP) had two types of effects on the longitudinal muscle of the mouse distal colon. At low concentrations (10(-8) M) VIP induced a contraction which seemed to be related to the production of prostaglandins as it was abolished after preincubation with indomethacin (10(-6) M). At higher concentrations (3 X 10(-8) and 10(-7) M) VIP induced relaxations which developed slowly and were related to stimulation of the adenylate cyclase activity of the smooth muscle cells. There is no evidence that VIP is the non-adrenergic, non-cholinergic transmitter released by electrical stimulation in this preparation and responsible for rapid relaxation of the smooth muscle.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenylyl Cyclases / metabolism
  • Animals
  • Colon / drug effects
  • Colon / enzymology
  • Colon / innervation*
  • In Vitro Techniques
  • Indomethacin / pharmacology
  • Mice
  • Muscle Contraction / drug effects
  • Muscle, Smooth / drug effects
  • Muscle, Smooth / enzymology
  • Neural Inhibition*
  • Neurotransmitter Agents*
  • Vasoactive Intestinal Peptide / antagonists & inhibitors
  • Vasoactive Intestinal Peptide / physiology*

Substances

  • Neurotransmitter Agents
  • Vasoactive Intestinal Peptide
  • Adenylyl Cyclases
  • Indomethacin