Successful combined targeting of B- and plasma cells in treatment refractory anti-NMDAR encephalitis

J Neuroimmunol. 2017 Nov 15:312:15-18. doi: 10.1016/j.jneuroim.2017.08.011. Epub 2017 Aug 25.

Abstract

We describe an extremely severe case of therapy refractory NMDA receptor encephalitis (NMDAe) in a 26-year-old woman. After rituximab, bilateral oophorectomy, repeated cycles of high dose methylprednisolone and plasma exchange, she received repeated cyclophosphamide, tocilizumab (interleukin-6 inhibitor) and finally bortezomib (plasma cell depleting drug) leading to remission after 204days in intensive care. Two years after disease onset her cognitive functions are still affected, but slowly improving and the cerebral atrophy has been partly reversed. The cerebrospinal fluid biomarker profile suggests an early synaptic/dendritic process, with subsequent neuroaxonal degeneration motivating aggressive treatment early on.

Keywords: Anti-N-methyl-d-aspartate receptor encephalitis; Bortezomib; Immunomodulatory therapy; Tocilizumab.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Anti-N-Methyl-D-Aspartate Receptor Encephalitis / cerebrospinal fluid
  • Anti-N-Methyl-D-Aspartate Receptor Encephalitis / diagnostic imaging
  • Anti-N-Methyl-D-Aspartate Receptor Encephalitis / physiopathology
  • Anti-N-Methyl-D-Aspartate Receptor Encephalitis / therapy*
  • B-Lymphocytes / drug effects
  • B-Lymphocytes / physiology*
  • Biomarkers / cerebrospinal fluid
  • Female
  • Humans
  • Immunologic Factors / therapeutic use
  • Longitudinal Studies
  • Magnetic Resonance Imaging
  • Plasma Cells / drug effects
  • Plasma Cells / physiology*
  • Plasma Exchange
  • Rituximab / therapeutic use

Substances

  • Biomarkers
  • Immunologic Factors
  • Rituximab