Sulfation can enhance antitumor activities of Artemisia sphaerocephala polysaccharide in vitro and vivo

Int J Biol Macromol. 2018 Feb;107(Pt A):502-511. doi: 10.1016/j.ijbiomac.2017.09.018. Epub 2017 Sep 8.

Abstract

In this study, a sulfated Artemisia sphaerocephala polysaccharide (ASPs) was prepared and its antitumor activity was evaluated in tumor cells and Hepatoma 22 (H22) tumor-bearing mice. In vitro experiments, ASPs significantly inhibited the growth of HepG2 and Hela cells with the IC50 values of 172.03 and 161.42μg/mL, respectively. Moreover, no direct cytotoxicity against mouse fibroblast L929 normal cells was observed in vitro. After oral administration for 12days, the tumor growth was significantly suppressed by ASPs at the doses of 200mg/kg (inhibition rate of 60.85%). Results of tumor histological morphology and cell cycle analysis showed that ASPs could arrest H22 cells at S phase and promote cell apoptosis. Additionally, immunohistochemical analysis demonstrated that ASPs caused the down-regulation of mutant p53 protein expression in a dose-dependent manner. Therefore, these findings proposed new insight into antitumor properties of sulfated polysaccharide as a promising agent in cancer treatment.

Keywords: Antitumor activity; Artemisia sphaerocephala polysaccharide; Sulfation.

MeSH terms

  • Animals
  • Apoptosis / drug effects
  • Artemisia / chemistry*
  • Carcinoma, Hepatocellular / drug therapy*
  • Carcinoma, Hepatocellular / pathology
  • Gene Expression Regulation, Neoplastic / drug effects
  • Hep G2 Cells
  • Humans
  • Liver Neoplasms / drug therapy*
  • Liver Neoplasms / pathology
  • Mice
  • Polysaccharides / administration & dosage*
  • Polysaccharides / chemistry
  • Sulfates / chemistry
  • Tumor Suppressor Protein p53 / genetics

Substances

  • Polysaccharides
  • Sulfates
  • TP53 protein, human
  • Tumor Suppressor Protein p53