[Molecular mechanisms of autophagy in regulating renal aging and interventional effects of Chinese herbal medicine]

Zhongguo Zhong Yao Za Zhi. 2016 Nov;41(21):3914-3918. doi: 10.4268/cjcmm20162105.
[Article in Chinese]

Abstract

Aging is the gradual functional recession of the living tissues or organs caused by a variety of genetic and environmental factors together. Autophagy is a process of degrading cytoplasmic components mediated by lysosomes in eukaryotic cells. Kidney is a typical target organ of aging. Autophagy regulates renal aging. Decrease in autophagy can accelerate renal aging,whereas,increase in autophagy can delay renal aging. During the process of regulating renal aging,the mammalian target of rapamycin (mTOR) and its related signaling pathways including the adenosine monophosphate activated protein kinase (AMPK)/mTOR,the phosphatidylinositol 3-kinase (PI3K)/ serine-threonine kinase(Akt)/mTOR,the AMPK/silent information regulation 1 (Sirt1) and transforming growth factor β (TGF-β) play the important roles in renal aging. Regulating the key signaling molecules in these pathways in vivo can control renal aging. Some Chinese herbal medicine (CHM) and their extracts with the effects of nourishing kidney or activating stasis, such as Cordyceps sinensis, curcumin and resveratrol have the beneficial effects on renal aging and/or autophagy. Therefore,revealing the pharmacological effects of CHM in anti-renal aging based on the molecular mechanisms of autophagy will become one of the development trends in the future study.

Keywords: Chinese herbal medicine; autophagy; mTOR signaling pathway; molecular mechanisms; renal aging.

Publication types

  • Review

MeSH terms

  • Adenylate Kinase / metabolism
  • Animals
  • Autophagy*
  • Drugs, Chinese Herbal / pharmacology*
  • Humans
  • Kidney / drug effects*
  • Phosphatidylinositol 3-Kinases / metabolism
  • Protein Serine-Threonine Kinases / metabolism
  • Proto-Oncogene Proteins c-akt / metabolism
  • Signal Transduction*
  • Sirtuin 1 / metabolism
  • TOR Serine-Threonine Kinases / metabolism
  • Transforming Growth Factor beta / metabolism

Substances

  • Drugs, Chinese Herbal
  • Transforming Growth Factor beta
  • Protein Serine-Threonine Kinases
  • Proto-Oncogene Proteins c-akt
  • TOR Serine-Threonine Kinases
  • Adenylate Kinase
  • Sirtuin 1