[Nonautonomous effects of oncogenic YAP in hepatocarcinogenesis]

Pathologe. 2017 Nov;38(Suppl 2):175-179. doi: 10.1007/s00292-017-0361-2.
[Article in German]

Abstract

Background: The transcriptional coactivator yes-associated protein (YAP) is a strong oncogene in liver cancer development.

Objectives: To investigate if and how YAP-induced paracrine-acting factors are regulated in hepatocytes and liver cancer cells.

Material and methods: Transcriptome analysis and proteomics of murine wildtype and YAP-transgenic hepatocytes were performed to identify paracrine-acting proteins. Molecular and biochemical techniques were used to examine the mechanisms of YAP-dependent gene regulation. Gene expression data from HCC (hepatocellular carcinoma) patients was evaluated.

Results: Several YAP-dependent, secreted factors (e. g. CXCL10, GDF15, PDGFB) were identified. YAP regulates these factors through transcription factors of the TEAD (TEA domain) protein family. Moreover, the dysregulation of the YAP-target genes is often associated with poor HCC patient prognosis.

Conclusions: YAP induces the expression of paracrine-acting factors that may affect the tumor microenvironment and therefore support carcinogenesis. This multicellular network could allow the development of novel and specific perturbation approaches.

Keywords: Carcinogenesis; Gene Expression Profiling; Hepatocellular carcinoma; Hepatocytes; Tumor microenvironment.

Publication types

  • Review

MeSH terms

  • Adaptor Proteins, Signal Transducing / metabolism*
  • Animals
  • Carcinogenesis / genetics*
  • Carcinoma, Hepatocellular / genetics*
  • Cell Cycle Proteins
  • Gene Expression Regulation, Neoplastic*
  • Humans
  • Liver Neoplasms / genetics*
  • Mice
  • Phosphoproteins / metabolism*
  • Transcription Factors
  • YAP-Signaling Proteins

Substances

  • Adaptor Proteins, Signal Transducing
  • Cell Cycle Proteins
  • Phosphoproteins
  • Transcription Factors
  • YAP-Signaling Proteins
  • YAP1 protein, human
  • Yap1 protein, mouse