Notch signaling pathway dampens tumor-infiltrating CD8+ T cells activity in patients with colorectal carcinoma

Biomed Pharmacother. 2018 Jan:97:535-542. doi: 10.1016/j.biopha.2017.10.143. Epub 2017 Nov 6.

Abstract

CD8+ T cells play critical role in controlling the metastasis and prognosis of cancer. Controversy remains as to the contribution of Notch signaling pathway in modulation of CD8+ T cells activity and development of tumorigenesis. Thus, the aim of the current study was to investigate the immunoregulatory role of Notch signaling pathway to peripheral and tumor-infiltrating CD8+ T cells in patients with colorectal carcinoma. A total of 46 patients with colorectal carcinoma and 20 health individuals were enrolled, and CD8+ T cells were purified from both peripheral bloods and carcinoma specimens. Cytolytic and noncytolytic functions of CD8+ T cells in response to Notch signaling inhibition were evaluated by measurements of lactate dehydrogenase release and proinflammatory cytokines production in both direct and indirect contact co-culture system to target HT29 cells. Cellular proliferation and inhibitory receptors expression in CD8+ T cells were also assessed by CCK-8 method and flow cytometry. There was no remarkable difference in percentage of CD8+ T cells between healthy individuals and patients with colorectal carcinoma. Notch1/2 and Hes1/5 mRNAs were elevated expressed in tumor-infiltrating CD8+ T cells in patients with colorectal carcinoma, however, did not correlated with tumor differentiation or stages. CD8+ T cells from healthy individuals presented stronger cytotoxicity, which was not affected by Notch signaling inhibitor. Inhibition of Notch signaling pathway not only promoted cytotoxicity of tumor-infiltrating CD8+ T cells, but also enhanced proinflammatory cytokines (including IFN-γ, TNF-α, IL-1β, IL-6, and IL-8) production by CD8+ T cells from patients with colorectal carcinoma. This process was accompanied by decreased expression of PD-1 in CD8+ T cells without influencing cellular proliferation. Our results indicated a potential immunosuppressive property of Notch signaling pathway, which dampened both cytolytic and noncytolytic functions of CD8+ T cells probably via induction of PD-1 in patients with colorectal carcinoma.

Keywords: CD8(+) T cells; Colorectal carcinoma; Notch signaling.

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • CD8-Positive T-Lymphocytes / immunology
  • CD8-Positive T-Lymphocytes / metabolism*
  • CD8-Positive T-Lymphocytes / pathology
  • Colorectal Neoplasms / immunology
  • Colorectal Neoplasms / metabolism*
  • Colorectal Neoplasms / pathology
  • Female
  • HT29 Cells
  • Humans
  • Lymphocytes, Tumor-Infiltrating / immunology
  • Lymphocytes, Tumor-Infiltrating / metabolism*
  • Lymphocytes, Tumor-Infiltrating / pathology
  • Male
  • Middle Aged
  • Receptor, Notch1 / immunology
  • Receptor, Notch1 / metabolism*
  • Receptor, Notch2 / immunology
  • Receptor, Notch2 / metabolism*
  • Signal Transduction / physiology*

Substances

  • NOTCH1 protein, human
  • NOTCH2 protein, human
  • Receptor, Notch1
  • Receptor, Notch2