Characterization of a Large Novel α-Globin Gene Cluster Deletion Causing α0-Thalassemia in a Chinese Family

Hemoglobin. 2017 Jul-Nov;41(4-6):297-299. doi: 10.1080/03630269.2017.1366919. Epub 2017 Nov 21.

Abstract

We report a large novel α-globin cluster deletion that we named - -PG (NG_000006.1: g.93628_542759del450131), in a Chinese family. This large deletion is approximately 450 kb long, spanning from upstream of the PolR3k gene at the 5' end to the RAB11FIP3 gene at the 3' end of chromosome 16p13.3. This deletion removes all the globin distal regulatory elements as well as the whole α-globin gene cluster. Patients with heterozygous - -PG/αα had red blood cell (RBC) indices consistent with α-thalassemia (α-thal) trait, but no apparent increase in a cancer tendency or mental disability, microcephaly, relative hypertelorism, unusual facies or genital anomalies.

Keywords: array comparative genomic hybridization (aCGH); multiplex ligation-dependent probe amplification (MLPA); α-Globin gene cluster; α-thalassemia (α-thal).

Publication types

  • Case Reports

MeSH terms

  • Adult
  • Base Sequence*
  • Family
  • Heterozygote*
  • Humans
  • Male
  • Multigene Family*
  • Sequence Deletion*
  • alpha-Globins / genetics*
  • alpha-Thalassemia / genetics*

Substances

  • alpha-Globins