Induction of apoptosis in human glioma cell lines of various grades through the ROS-mediated mitochondrial pathway and caspase activation by Rhaponticum carthamoides transformed root extract

Mol Cell Biochem. 2018 Aug;445(1-2):89-97. doi: 10.1007/s11010-017-3254-z. Epub 2017 Dec 14.

Abstract

The present study is the first investigation of the inhibitory effect of Rhaponticum carthamoides transformed roots (TR) extract on the proliferation of grade II and III human glioma cells. TR extract showed the cytotoxic effect and inhibited the colony formation of both glioma cell lines in dose-dependent manner. The root extract induced apoptosis by increasing of the reactive oxygen species (about threefold compared to the control cells) leading to a disruption of mitochondrial membrane potential. Additionally, the mRNA levels of the apoptotic factors such as Bax, Tp53, caspase-3, and caspase-9 were observed to increase. These results indicate that the TR extract possesses anticancer activity by inhibiting glioma cell proliferation and inducing apoptotic cell death, and may be used as a promising anticancer agent.

Keywords: Apoptosis; Bax; Bcl-2; Caffeic acid derivatives; Caspase-3; Caspase-9 gene expressions; Human glioma cells; Rhaponticum carthamoides; Tp53; Transformed roots.

MeSH terms

  • Antineoplastic Agents, Phytogenic / pharmacology
  • Brain Neoplasms / enzymology
  • Brain Neoplasms / metabolism
  • Brain Neoplasms / pathology*
  • Caspase 3 / genetics
  • Caspase 3 / metabolism*
  • Caspase 9 / genetics
  • Caspase 9 / metabolism*
  • Cell Line, Tumor
  • Cell Proliferation / drug effects
  • Enzyme Activation
  • Glioma / enzymology
  • Glioma / metabolism
  • Glioma / pathology*
  • Humans
  • Leuzea / chemistry*
  • Leuzea / growth & development
  • Leuzea / metabolism
  • Membrane Potential, Mitochondrial / drug effects*
  • Middle Aged
  • Plant Extracts / pharmacology*
  • Plant Roots / chemistry*
  • Plant Roots / growth & development
  • Plant Roots / metabolism
  • Reactive Oxygen Species / metabolism*
  • Tumor Suppressor Protein p53 / drug effects
  • bcl-2-Associated X Protein / genetics

Substances

  • Antineoplastic Agents, Phytogenic
  • Plant Extracts
  • Reactive Oxygen Species
  • TP53 protein, human
  • Tumor Suppressor Protein p53
  • bcl-2-Associated X Protein
  • Caspase 3
  • Caspase 9