A light and electron microscopical study of B-50 (GAP-43) in human intramuscular nerve and neuromuscular junctions during development

J Neurol Sci. 1989 Feb;89(2-3):301-11. doi: 10.1016/0022-510x(89)90031-2.

Abstract

The growth associated protein B-50 (GAP-43) is demonstrated in human intramuscular nerves and neuromuscular junctions by light microscopy and electron microscopy. Nearly all fetal endplates are shown to be immunoreactive for B-50. The percentage of B-50 positive endplates decreases significantly during the peri- and neonatal period, but in children and adults a low percentage of B-50 positive endplates remains present. These data indicate that also in the human peripheral nervous system the expression of B-50 is developmentally regulated.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aging / metabolism*
  • Antibodies, Monoclonal
  • Child
  • Fetus / metabolism*
  • GAP-43 Protein
  • Humans
  • Membrane Proteins / metabolism*
  • Microscopy, Electron
  • Muscle Development
  • Muscles / innervation*
  • Muscles / ultrastructure
  • Nerve Tissue Proteins / metabolism*
  • Neuromuscular Junction / metabolism*
  • Neuromuscular Junction / physiology
  • Neuromuscular Junction / ultrastructure

Substances

  • Antibodies, Monoclonal
  • GAP-43 Protein
  • Membrane Proteins
  • Nerve Tissue Proteins