Insulin Resistance and β-Cell Dysfunction in Relation to Cardiometabolic Risk Patterns

J Clin Endocrinol Metab. 2018 Jun 1;103(6):2207-2215. doi: 10.1210/jc.2017-02584.

Abstract

Context: Insulin resistance (IR) and β-cell dysfunction are two major defects synergistically inducing the development of diabetes and related cardiometabolic disorders.

Objective: To investigate the independent and joint associations of IR and β-cell dysfunction with the prevalence of multiple cardiometabolic disorders, including obesity, central obesity, diabetes, dyslipidemia, and hypertension.

Design and settings: A nationally representative population of 93,690 Chinese adults.

Main outcome measures: IR and β-cell dysfunction were assessed by the homeostasis model assessment of IR (HOMA-IR) and of β-cell function (HOMA-B), respectively.

Results: High HOMA-IR was independently associated with high prevalence of all estimated cardiometabolic disorders, whereas low HOMA-B was independently associated with high prevalence of diabetes, dyslipidemia, and hypertension but low prevalence of obesity and central obesity. When examined jointly, the associations of HOMA-IR and HOMA-B with multiple cardiometabolic disorders showed different patterns with varying magnitudes. The strongest joint associations were observed for diabetes, with low HOMA-B associated with high prevalence of diabetes regardless of HOMA-IR; joint associations with dyslipidemia and hypertension prevalence appeared to be additive and had moderate changing trends; and low HOMA-B was not associated with high prevalence of obesity or central obesity unless combined with high HOMA-IR.

Conclusion: IR was associated with more prevalent cardiometabolic disorders than was β-cell dysfunction, and combinations of IR and β-cell dysfunction showed distinct relations with cardiometabolic risk patterns in Chinese adults.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Blood Glucose*
  • Body Mass Index
  • Diabetes Mellitus, Type 2 / epidemiology*
  • Diabetes Mellitus, Type 2 / etiology
  • Diabetes Mellitus, Type 2 / metabolism
  • Dyslipidemias / epidemiology*
  • Dyslipidemias / etiology
  • Dyslipidemias / metabolism
  • Female
  • Glucose Tolerance Test
  • Humans
  • Hypertension / epidemiology*
  • Hypertension / etiology
  • Hypertension / metabolism
  • Insulin Resistance / physiology*
  • Insulin-Secreting Cells / metabolism*
  • Male
  • Middle Aged
  • Obesity / epidemiology*
  • Obesity / etiology
  • Obesity / metabolism
  • Prevalence
  • Risk Factors

Substances

  • Blood Glucose