Metabolic reprogramming of host cells upon bacterial infection: Why shift to a Warburg-like metabolism?

FEBS J. 2018 Jun;285(12):2146-2160. doi: 10.1111/febs.14446. Epub 2018 Apr 14.

Abstract

The finding that the Warburg effect observed in proliferating cancer cells is also observed during immune responses renewed the interest in the study of metabolic reprogramming of immune cells, a field of investigation called immunometabolism. However, the specific mechanisms and processes underlying metabolic changes of host cells upon bacterial infection remain poorly understood. Several recent reports have reported that mammalian cells infected with intracellular bacteria have an altered metabolism that resembles the Warburg effect seen in cancer cells. In this Review, we will summarize current knowledge on metabolic reprogramming and discuss putative causes underlying the preferential remodelling of host cells to Warburg-like metabolic programs during infection by intracellular bacteria.

Keywords: OXPHOS; Warburg-like metabolism; bacterial infection; glycolysis; mitochondria.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Animals
  • Bacteria / growth & development
  • Bacteria / pathogenicity
  • Cell Differentiation
  • Cell Proliferation
  • Cellular Reprogramming / drug effects
  • Glycolysis / genetics*
  • Host-Pathogen Interactions*
  • Humans
  • Interleukin-1beta / biosynthesis
  • Interleukin-1beta / immunology
  • Lipopolysaccharides / pharmacology
  • Macrophages / drug effects
  • Macrophages / immunology
  • Macrophages / metabolism*
  • Macrophages / microbiology
  • Mitochondria / drug effects
  • Mitochondria / immunology
  • Mitochondria / metabolism*
  • Mitochondria / microbiology
  • Oxidative Phosphorylation / drug effects
  • T-Lymphocytes / drug effects
  • T-Lymphocytes / immunology
  • T-Lymphocytes / metabolism*
  • T-Lymphocytes / microbiology

Substances

  • Interleukin-1beta
  • Lipopolysaccharides