Increased platelet aggregation and release reaction in myotonic dystrophy

J Neurol Sci. 1988 Apr;84(2-3):239-46. doi: 10.1016/0022-510x(88)90128-1.

Abstract

Platelet aggregation (PA) induced by (-)-epinephrine and adenosine diphosphate (ADP) was studied in 16 patients with myotonic dystrophy (MyD) and 14 healthy subjects. Plasma beta-thromboglobulin level (beta-TG), a useful marker of in vivo platelet release reaction, as well as in vitro 5-[14C]hydroxytryptamine (5-HT) release, were also studied. The extent of PA induced by (-)-epinephrine at 1 and 3 min and by ADP at 3 min was significantly higher in the patients than in controls. Plasma beta-TG and ADP- or epinephrine-induced platelet 5-HT release were also increased in MyD patients. These results suggest that an abnormality in release as well as in alpha 2-receptor functioning occurs in the platelets of MyD patients. The relation of this abnormality to changes in Ca2+ fluxes through the platelet membrane is discussed.

MeSH terms

  • Adenosine Diphosphate / pharmacology*
  • Adult
  • Epinephrine / pharmacology*
  • Female
  • Humans
  • Male
  • Myotonic Dystrophy / blood*
  • Myotonic Dystrophy / physiopathology
  • Platelet Aggregation / drug effects*
  • Serotonin / physiology
  • beta-Thromboglobulin / physiology

Substances

  • beta-Thromboglobulin
  • Serotonin
  • Adenosine Diphosphate
  • Epinephrine